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Scientifica 22 BianY.LeiY.WangC.WangJ.WangL.LiuL.et al (2015)

These effects are seen primarily at low sublethal acrolein concentrations, and may be due to hormesis described for other toxins, wherein hormesis-stimulating compounds initiate an adaptive response that confers protection against harmful doses of the same or similar agents

It was an even more effective blocker of the channel type affected in the two sisters. Further research by the WashU investigators showed that norfluoxetine, a breakdown product of fluoxetine that accumulates in fluoxetine users, had a remarkably targeted ability to block the gain of function channels produced by the harmful genetic variation

doi: 10.2337/db18-0638