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doi: 10.1093/jnci/djt181
Rasmussen J, Dun M, Lund AM, et al
Unbiased transcriptomic and metabolomic analyses showed that chronic 2 -adrenergic receptor stimulation caused metabolic reprogramming of skeletal muscle characterized by enhanced glucose utilization
As previously mentioned, CPT1 may be allosterically inhibited by malonyl-CoA, a product of ACC (Figure 3)
