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References Forloni G, et al
NivoliezAVeisseirePAlaterreEDaussetCBaptisteFCamarsOet al
7D) could be due to the loss of function mutations in the HLA-A allele in some clones within the tumour, making them unresponsive to neoepitope-driven T-cell activation, thereby contributing to relapse of the disease in LS patient II.2
