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63 (19), e1801390

These discrepancies may be explained in part by the fact that while in most systems versions of CSD mimic the effects of endogenous CAV-1, in some systems they act as competitors of the activity of endogenous CAV-1 31,32,34,35,36

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Future Research Directions for Longevity Peptide Therapeutics MOTS-c represents an intriguing example of mitochondrial signaling with measurable human biomarker data