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Through these mechanisms, PSORI-CM02 effectively disrupts the oxidative stressinflammationangiogenesis axis, contributing to its therapeutic potential in managing psoriasis (Lu et al
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Cellular GSSG accumulation, as opposed to GSH reduction, rendered hepatocytes susceptible to TNF cytotoxicity Increased GSSG/GSG ratios in response to H 2 O 2 exposure can stem from both GSH reduction and GSSG accumulation (data not shown)
