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Carvalho-Wells et al., Figure 3 The mechanism for hypertriglyceridemia in APOE4 may involve its stronger binding to VLDL, which decreases lipoprotein lipase mediated lipolysis (Li et al., APOE4 is through the sequestration of apoE proteins on the hepatic cell surface
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A great amount of Th17cell phenotype has been linked to ASD pathogenesis (Moaaz et al., 2019)
Beige adipocytesanother type of UCP1-expressing adipocytesare dramatically induced in certain depots of white adipose tissue (WAT) by long-term cold exposure, facilitating thermoregulation and energy homeostasis [2]
