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However, FABP1 protein level was decreased in a dose-dependent manner in L02 cells overexpressing GSTA1 (Figure 6G)

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Emerging evidence highlights blood-brain barrier (BBB) dysfunction as a critical factor in the pathophysiology of schizophrenia, potentially triggering neuroinflammatory cascades, aberrant neurotransmission and structural remodeling that contribute to symptom onset and progression (5,6)

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