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doi: 10.1101/cshperspect.a000398 11 MaloneMBjarnsholtTMcBainAJJamesGAStoodleyPLeaperDet al
Iron accumulation drives fibrosis, senescence and the senescence-associated secretory phenotype
The prophylactic administration of BB in the murine model of UC (BB post-UC) reduced histological damage and prevented the expression of IL-1 by 61.12%, IL-6 by 94.70%, and TNF- by 68.88%, increased the expression of INF- by 177.06%, IL-4 by 541.36% and IL-10 by 531.97%, concerning the control group that underwent UC induction (H 2 O post-UC) (Table 5)
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