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Although GSH failed to restore 5-hydroxytryptamine (5-HT/serotonin) levels in embryos treated with 10 M CdCl2, the Cd-induced suppression of heart rate was reversed by the serotonergic (5-HTergic) modulators quipazine and fluoxetine, suggesting the involvement of the 5-HT system in Cd-induced cardiotoxicity

Results Despite impaired cardiolipin remodelling, pancreatic islets from Taz-KD mice maintained insulin secretion, supported by compensatory mechanisms such as increased glucose uptake, expanded mitochondrial volume and increased metabolic parameters

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