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In summary, vitamin D exerts renoprotective effects on renal fibrosis by suppressing profibrotic signaling, modulating multiple molecular pathways, and stabilizing the renal microenvironment
Evidence includes observations of reduced oxidative stress markers, improved mitochondrial parameters, and decreased indicators of DNA damage
In addition, Caveolin-1 (Cav-1), a 21-kDa integral plasma membrane protein is a well-known negative regulator of eNOS activity in vascular endothelial cells 1722
Silencing ER triggered ferroptosis, which was attenuated by ferroptosis inhibitor Ferrostatin-1, and was enhanced by ferroptosis inducer Erastin
