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Proteolytic stability is often given as an explanation why BPC-157 was under investigation in several ways of administration in animal researches, and why this compound is often mentioned as resistant to degradation in acidic environment, where other peptides decompose easily

By combining GLS1 inhibition (e.g., CB-839) and NQO1 -bioactivatable drugs (e.g., -lap, ARQ761), we exploit the reliance of PDA on glutamine for redox balance, as well as the tumor-selective overexpression of NQO1 through the use of a unique agent that is bioactivated to induce cell death

Ryanodine receptor leak mediated by caspase-8 activation leads to left ventricular injury after myocardial ischemia-reperfusion

Lutgens E, Atzler D, Dring Y, Duchene J, Steffens S, Weber C