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Gut 51:842848
Anti-neuroinflammation effect of ginsenoside Rbl in a rat model of Alzheimer disease
Importantly, there is very little direct experimental evidence in support of this hypothesis, which is based on the assumption that the neurotoxicity is caused by similar mechanisms as described for hepatotoxicity [183]
Here, we report on such a dual engineering approach, where the REW substitutions in IgG1 resulted in favorable pharmacokinetics in human FcRn expressing mice, combined with increased ability to activate the classical complement pathway in an on-target manner
