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Description
These effects are typically minimal and resolve without intervention
Studies with Egyptian children and adolescents, for example, show that the null genotype of conferred a 4.2-fold increased risk for the occurrence of DM, and in this case, associations with some biochemical variables and laboratory data were also observed (lipid profile and HbA1c)
However, RNAi-mediated SLC7A11 gene knockout (xCTKD) can significantly reverse this resistance and increase the sensitivity of glioma cells to sulfasalazine [53]
C9orf72-mediated ALS and FTD: multiple pathways to disease
