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At present, there are several crosstalk mechanisms between classical regulatory genes of ferroptosis in ovarian diseases ( Figure 2 )
The HPV16 E7 oncoprotein directly suppresses the cyclic GMP-AMP synthase (cGAS)-stimulator of interferon genes (STING) signaling pathway, significantly reducing the expression of genes that encode type I interferon and pro-inflammatory proteins, hence facilitating immune evasion in several HPV-related tumors in murine models (Luo et al., 2020)
Data analysis: ARM, MM, HM, MJL, and SG
Oxidative stress and reactive oxygen species: a review of their role in ocular disease
