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This suggests that a substantial fraction of OCR in steady state is due to oxidation of FAs 37 as shown in a previous study, and that the source of the FAs in our model is through lipophagy

A significant observation in our data is that people with autism and low plasma free carnitine are metabolically diverse and there should therefore be no a priori expectation that all individuals with autism and low free carnitine will have similar autism-related pathobiology or identical clinical responses to carnitine supplementation

11,20,21 Additionally, non-pharmacological approaches, including lifestyle modifications, are being explored to enhance patient outcomes and quality of life

However, portion control is key to prevent unnecessary weight gain