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The multifactorial nature of DED encompasses a number of interconnected underlying pathologies, including a loss of homeostasis, instability and hyperosmolarity of the tears, and the induction and propagation of detrimental inflammatory responses in the eyes, which finally results in the development of neurosensory dysfunction and visual disruption [1,5]
Sphingosine-1-phosphate induces an antiinflammatory phenotype in macrophages
