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Abstract Idiopathic pulmonary fibrosis (IPF), the most common form of idiopathic interstitial pneumonia, is a progressive, irreversible, and typically lethal disease characterized by an abnormal fibrotic response involving vast areas of the lungs

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Some studies propose that macrophage factor IL-1 and Th17 cytokine IL-17A may negatively impact GR function by upregulating the expression of GR subtypes, with GR being the primary structure exerting effects (335, 336)