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assessed 101 HCV patients and demonstrated a significant correlation between steatosis and HCV RNA level in patients infected with HCV-3, but not in those infected with HCV-1, providing evidence that steatosis is the morphological expression of a viral cytopathic effect of HCV-3.[27] Furthermore, in patients with HCV cirrhosis, genotype 3 has been independently associated with an increased risk of HCC[28] (Figure 1)

doi: 10.1194/jlr.R400013-JLR200 48 GlomsetJ

Further highlighting the link between insulin resistance and postprandial lipid abnormalities, a recent study found that insulin resistant, abdominally obese adults had higher postprandial TG concentrations than abdominally obese adults without insulin resistance and non - abdominally obese controls (61, 86)

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