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Description
Although early NAC treatment is beneficial in limiting the injury, continuous NAC treatment well beyond the injury phase can delay regeneration [72] mainly through impairment of mitochondrial biogenesis, an adaptive response whose role is to maintain or restore homeostasis following mitochondrial dysfunction [71]
In the spotlight of neurochemists is ketimine reductase (-crystallin), which acts as a thyroid hormone-binding protein
What is Glutathione best known for
Source: Xu et al., Nature (2025)
