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Mechanism studies showed that p53-induced miR-145a inhibited osteogenesis by targeting core binding factor beta (Cbfb) [140]
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For atoms within QM, split valence polarization atomic basis sets (SVP) 52 and the hybrid B3LYP functional 53,54,55,56 have been employed
Similarly, enforced GLUT3 expression in OT1 T cells promoted a sustained memory phenotype after repeated antigen stimulation in vitro and enhanced protection after tumor rechallenge in vivo 52
