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Affects multiple atherosclerotic processes including endothelial function, oxidative stress, foam cell formation, inflammatory response (its anti-inflammatory), platelet aggregation and plaque rupture (it causes plaque regression) [134, 135]

These microbial shifts coincide with elevated SCFA levels and increased expression of UCP1, CPT1B, and SIRT1 in adipose depots, indicating that AX promotes thermogenesis through a gut microbiotametaboliteUCP1 regulatory axis that integrates intestinal and systemic energy homeostasis [73,75,76]

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