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Similarly, shortly after CNS axon injury in vitro and in vivo, lesioned neurons exhibit dendritic spine loss, and receive heightened presynaptic excitatory signals along with diminished inhibitory inputs [42]
Surprisingly, there is abundant experimental evidence that CK1, PKC, and JAK3 promote eryptosis, while in non-erythrocytic cells, these kinases are mostly antiapoptotic
Stamatoyannopoulos J
Notably, additional studies should address tumor heterogeneity, complex interactions within the TME, and mechanisms of therapeutic resistance
