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From a therapeutic perspective, understanding the molecular mechanisms that allow functional adaptations of ENS signaling during tumorigenesis represents an exciting challenge

found that microcystin-LR is selectively absorbed by macrophages in lung tissue and binds to GRP78, attenuating M2 polarization of macrophages by reducing p-PERK, p-IRE1, and cleaved ATF6 (178)

Goodyer, C
