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In the selenite-induced cataract model, the extract likely mitigated oxidative stress by scavenging reactive oxygen species, thereby preventing protein damage and maintaining the levels of essential lens proteins such as -A crystallin and aquaporin 0

However, increased expression of this gene was observed in cells pretreated with 5 and 10 M CBD and exposed to 6-OHDA, indicating that the neuroprotective properties of CBD are mediated through the Nrf2 signaling pathway

Intriguingly however, in glr1 cells, we observed almost no detectable NADP oxidation at all, except for a slight response of the NAPstar3 probe to the highest diamide concentration (5 mM) ( Figure 5C,E )