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Description
Emerging evidence suggests that modulation of SCFA production may offer therapeutic avenues for neuroinflammatory and neurodegenerative conditions
While no direct interactions have been documented, individual responses can vary based on personal health conditions, dosage, and duration of use
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Therefore, they constitute a platform for a complex set of interactions involving drug metabolism, protein haptenation, modulation of the inflammatory response, and/or generation of danger signals with implications in drug hypersensitivity reactions
