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2013;11(1, article 173) doi: 10.1186/1479-5876-11-173

AIP in particular presents an attractive opportunity for liver-targeted gene therapy because only a few functional hepatocytes expressing HMBS are needed to metabolize ALA/PBG in order to avert acute attacks

We therefore propose a simple, testable framework: CSCs from embryonal/embryonic-like tumors may depend on ferritin chiefly to buffer iron and preserve stemness under fluctuating microenvironments, whereas CSCs from differentiated epithelial tumors may exploit iron in specialized metabolic or redox processes, In this context, ferritin loss can trigger stress-adaptive programs (proliferation, migration, EMT) that paradoxically increase aggressiveness [123,124,125]

Claudin-1 and -2: Novel Integral Membrane Proteins Localizing At Tight Junctions With No Sequence Similarity to Occludin