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Studies examine: VEGFR2 activation pathways and intracellular adaptor protein recruitment Focal adhesion kinase (FAK) phosphorylation mechanisms Paxillin recruitment and integrin-actin cytoskeleton interactions Endothelial cell proliferation and migration assays in vitro Research models study the combined effects of VEGFR2-mediated proliferation and FAK-paxillin-mediated cellular motility in angiogenesis pathway investigations

More recent work from the Zagreb group has explored BPC-157 in the context of the gut-brain axis, investigating how its gastrointestinal effects may relate to broader systemic and neurological outcomes a line of inquiry that reflects growing scientific interest in enteric-central nervous system communication