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Released AIF translocates to the nucleus where it promotes chromatin condensation and DNA fragmentation

Conversely, inflammatory phyla were notably expanded: Pseudomonadota (Proteobacteria) showed extreme enrichment, suggesting gut inflammation and barrier disruption, while Campylobacterota was significantly increased, consistent with pathobiont overgrowth

Since the inhibition of ASK1 (apoptosis signal-regulation kinase 1) in NLRP3 (NOD-like receptor thermal protein domain associated protein 3) mutant mice can reduce liver fibrosis, hepatocyte death, and liver TNF- expression, it suggests the application prospects of ASK1 inhibitor selonsertib in liver diseases

Sarker SA, McCallin S, Barretto C, et al