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however, the gliP deletion strain showed no difference in virulence towards completely immunosuppressed mice in a low-dose model compared with wild strain [26], thus further supporting the conclusion that gliotoxin is not required for the pathogenicity of A

However, when GSH-PEG liposomes were used to deliver an anti-amyloid single domain antibody fragment (VHH-pa2H) to the brain of mice, no increase in brain delivery was observed when compared with VHH-pa2H administered alone [48]

doi: 10.3164/jcbn.19-87

In the in vitro experiments, construction of the HK-2 cell ferroptosis model demonstrated that RGSH could significantly increase cell viability and significantly increase the lipid oxide level in cells, which inhibited cell apoptosis