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confirmed that TFAP2C regulated GPx1 expression by directly binding to the GPx1 promoter, which contains an AP-2 regulatory region, and the methylation of the CpG island in the GPx1 promoter region prevented the binding of TFAP2C by encompassing the AP-2 regulatory region [81]

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To construct the pRF HK2 5 UTR plasmid, the 5 UTR of mouse HK2 was amplified using Pfu polymerase (SolGent)

[4] Oral glucose tolerance evaluation across the pooled trial population indicated that, in contrast to intact hGH, AOD-9604 did not appear to interact with carbohydrate metabolism negatively