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Moreover, excessive APAP increases hepatic CXCL16 levels, CXCL16 deficiency significantly reduces the production of pro-inflammatory cytokines TNF- and IL-6, and inhibits neutrophils migration to the site of injury, other chemokines CXCL1 and CXCL2 expression are also diminished, suggesting that CXCL16 may be involved in neutrophil infiltration in APAP hepatotoxicity [38]

doi: 10.3390/toxics6030047

Increased adipocyte O2 consumption triggers HIF-1, causing inflammation and insulin resistance in obesity

Notably, close attention should be paid to the methods that can improve ferroptosis resistance of ectopic endometrial cells in the ovary while reducing the iron damage to adjacent ovarian granulosa cells and oocytes