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Sodium butyrate inhibits high cholesterol-induced neuronal amyloidogenesis by modulating NRF2 stabilization-mediated ROS levels: involvement of NOX2 and SOD1

Beyond well-established compounds like THC and CBD, the quest for novel cannabinoids widens the scope of potential treatments

Upregulation and release of S100 to the extracellular matrix could also initiate interaction with RAGE that eventually activates NF-B, inducing the production of pro-inflammatory cytokines and migration of neutrophils, monocytes, and macrophages (Xia et al., 2018)

In this report, we test this hypothesis by treating the HRAS G12V -transformed ovarian epithelial cells, mutant KRAS -harboring pancreatic and colon cancer cells of mouse and human origins, as well as cancer xenografts, with diphenyleneiodonium (DPI) and buthionine sulfoximine (BSO) combination, which inhibit NOX activity and glutathione biosynthesis, respectively