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Draining glutathione, the other hallmark of both diseases, produced nearly identical results through a completely separate route, suggesting the two factors converge on the same danger zone

179,180 Nonetheless, through the integration of multi-omics analysis and validation both in vitro and in vivo in NSCLC, a recent study disclosed that intracellular lactate drives extracellular lipolysis and FFA release via non-histone lactylation of apolipoprotein C-II (APOC2), bringing about immunotherapy resistance