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Description
In mice, AX increased fat utilization and running time to exhaustion by enhancing mitochondrial -oxidation and preventing oxidative modification of carnitine palmitoyltransferase I (CPT-I), a key enzyme in lipid metabolism [34]
The mitochondrial membrane permeabilization results in the release of the proapoptotic factors from the mitochondrial intermembrane space, cytochrome c [194], AIF [195], and endonuclease G [196], to the cytosol
As expected, at the end of the experiment, HFD mice gained 20% more BW than STD mice (Supplementary Figure S1A), together with increased heart weight (HW) and serum NEFA (Supplementary Figures S1BD)
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