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glutathione oxalates The SOX4/EZH2/SLC7A11 signaling axis mediates ferroptosis in calcium oxalate crystal deposition-induced kidney injury | Journal of Translational Medicine Oxalates - High and Low

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N/S co-doping introduced C = O, CN, and other functional groups, transforming the energy storage mechanism from electric double-layer capacitance (EDLC) toward reversible surface adsorption/redox pseudocapacitance, thereby maintaining capacity at high rates

glutathione oxalates The SOX4/EZH2/SLC7A11 signaling axis mediates ferroptosis in calcium oxalate crystal deposition-induced kidney injury | Journal of Translational Medicine Oxalates - High and Low

Acknowledgements The authors thank all those who contributed to this article, including Figdraw

glutathione oxalates The SOX4/EZH2/SLC7A11 signaling axis mediates ferroptosis in calcium oxalate crystal deposition-induced kidney injury | Journal of Translational Medicine Oxalates - High and Low

The slopes for CON and SZ plasma samples were slightly negative showing an overall decrease in GSH level with increase in storage time (Figure 1)

glutathione oxalates The SOX4/EZH2/SLC7A11 signaling axis mediates ferroptosis in calcium oxalate crystal deposition-induced kidney injury | Journal of Translational Medicine Oxalates - High and Low

Chelala L, OConnor EE, Barker PB, Zeffiro TA

glutathione oxalates The SOX4/EZH2/SLC7A11 signaling axis mediates ferroptosis in calcium oxalate crystal deposition-induced kidney injury | Journal of Translational Medicine Oxalates - High and Low

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