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Furthermore, in a Drosophila model of PD, alterations in the tryptophan metabolic pathway were in line with the overexpression of -Syn in the disease (Luan et al., 2015
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Associations between MTHFR C677T and MS vary across populations, some case-control studies identify risk signals, while others do not, supporting a 'substrate-limitation' model where genetic predisposition interacts with B-vitamin status to constrain DNMT activity and stabilize a proinflammatory methylation landscape (7,142,143)
