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Interestingly, similar to previous reports that myristoylation recruits FSP1 to the plasmalemma where it functions as an oxidoreductase is necessary to confer ferroptosis resistance (Bersuker et al

While apoptosis involves caspase activation, chromatin fragmentation and the phagocytosis of cell bodies, which minimizes inflammation, necrosis is defined by cell swelling and lysis with release of intracellular components, which elicits an inflammatory response [9]

Given its excessiveness, its highly likely that it wont remain in this form once it makes it into print