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Conjugates formed between glutathione and toxic metabolites are further metabolised into cysteinyl-s-conjugates, which have long half-lives in circulation

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As exhibited in a prior study, increased MAM formation elevated cytoplasmic Ca 2+ levels, which promoted the phosphorylation of serum response factor (SRF) and its binding to the vascular endothelial growth factor receptor 2 (VEGFR2) promoter, producing VEGFR2 and thereby promoting inflammatory angiogenesis

chronic or excessive activation may disrupt metabolic homeostasis or trigger off-target effects