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In a zebrafish model, compared with C3-carnitine, C18-carnitine and C18:1-carnitine can induce the inhibition of OXPHOS, interfere with the energy supply of cardiomyocytes, and lead to a significant impairment of cardiac systolic function, which manifests as a decreased heart rate and reduced shortening of the atrial and ventricular fractions [21]

This can lead to direct damage to cellular organelles including mitochondria

Thus, these two ASD-related synaptic proteins directly interact via SH3-binding domains and indirectly through binding to actin, determining the crosstalk between PMS and ADNP syndrome

In contrast, in the adult human, intravenously administered acetylcarnitine as a bolus (500 mg) led to a rapid increase in plasma concentration followed by a progressive decline reaching the base value in 12 hours