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Despite its crucial role in HCC progression, effective therapies directly targeting YAP1 remain challenging, underscoring the need to explore the regulatory mechanisms underlying its aberrant expression and activation
Discussion of the literature and results of a consecutive series of surgically treated patients
Animal studies have suggested that Apelin-13 attenuates secondary injury after TBI, and exerts a neuroprotective effect by suppressing autophagy, preventing BBB disruption, and alleviating brain edema [154]
doi: 10.1042/bj2660453
