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Blocking the mitochondrial apoptotic pathway preserves motor neuron viability and function in a mouse model of amyotrophic lateral sclerosis

Furthermore, mice with cardiac-specific overexpression of mutated human B-crystallin (hR120GCryAB) displayed increases in GSH levels and in activities of GR, glucose-6-phosphate dehydrogenase (G6PD), catalase, and GPx (indicative of reductive stress), which led to the development of cardiac hypertrophy and heart failure in these mice (77, 78)

Immediate discontinuation and treatment may be required if these reactions occur

Absorbance at OD 415 was measured and GSH concentration was determined by constructing a standard curve using GSH solutions provided by the manufacturer