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NO activates IRP1, which halts the translation of FPN1 and ferritin and promotes that of DMT1 and TfR1, favoring the increment of the cellular labile iron pool, which triggers the Fenton reaction and ferroptosis-mediated neuronal cell death [80, 310, 311]
Only around three small pilot studies exist
As the glycolytic pathway is impaired, pyruvate is most likely generated from amino acids such as serine, alanine and cysteine
