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Therefore, evaluating the potential anti-cancer effects of antioxidants on an individualized basis is crucial

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Abstract Damage-associated molecular patterns (DAMPs) are endogenous danger signal molecules released by damaged, stressed or dead cells that bind to pattern recognition receptors (PRRs), activating immune responses and inflammatory signaling pathways to play critical regulatory roles in various pathophysiological processes
The underlying mechanism of how GSH modulate the complex crosstalk between ferroptosis and autophagy remains unclear
