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Abstract Formation of the reactive amodiaquine quinoneimine (AQ-QI) and N -desethylamodiaquine quinoneimine (DEAQ-QI) plays an important role in the toxicity of the anti-malaria drug amodiaquine (AQ)

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The calcium regulation is imparted by the associated calmodulin subunits, thus explaining how vasodilators such as acetylcholine and bradykinin effect smooth muscle relaxation as a consequence of increasing intracellular endothelial cell calcium levels

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