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Chondrocytes that lack G6PDH are NADPH-insufficient and, in response to the impaired GSSG recycling and reduced protein folding, they activate the UPR and protein degradation to avoid proteotoxicity and cell death

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As the realization of the glycocalyxs primary role in preventing and/or ameliorating cardiovascular pathologies continues to emerge, many therapies targeting either increased biosynthesis (adsorption) or decreased degradation are currently in development to target the structure for protection or restoration

The PERK-ATF4-CHOP pathway is a central signaling cascade activated in response to ER stress, serving as the cells primary reaction to dysfunction and playing a crucial role in cellular survival