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Importantly, we further identified HO-1 mitochondrial translocation as a key mechanism for oxidative stress-induced mitochondrial iron overload and ROS accumulation

Emerging evidence underscores the key role of immune metabolic dysregulation in SLE, revealing how metabolic reprogramming during immune cell activation influences disease development and progression

Mechanisms linking environmental agents to the development of Autoimmune Diseases Oxidative stress-mediated disturbance of Nrf2, induction of NF-kB, PARP1, and NLRP3 can cause activation of both innate and adaptive immune systems, resulting in pro-inflammatory cytokines and production of autoantibodies, leading to tissue damage in Autoimmune disease

Thirty minutes later, the monolayer was washed three times with 20 mM Tris-HCl buffer, pH 7.4, containing 0.15 M sodium chloride (TBS)