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Description
Semaglutide's weight reduction is driven by appetite suppression and metabolic changes through GLP-1 receptor activation
Figure 1 Initiated by dysbiosis, harmful gut-derived products including LPS and FFAs translocate into the liver via the portal circulation
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Importantly, this redox-supportive role intersects with the glutamateglutamine cycle, a central regulator of neurotransmitter balance and oxidative homeostasis in the central nervous system [124]
