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The first group consists of DNMT inhibitors, which are hypomethylating (or demethylating) agents that can restore the expression of genes silenced by hypermethylation of their promoters

Clinically, strategies aimed at modulating EAAC1-mediated cysteine transport and boosting endogenous GSH synthesis may hold promise for mitigating post-ischemic neurodegeneration and cognitive deficits associated with vascular dementia

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Additionally, the hypoxic condition induced by anemia may worsen oxidative stress via pro-oxidant changes, including altered cellular metabolism, increased catecholamine metabolism, and leukocyte activation, thereby leading to increased free radical production and oxidative stress [41]