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Description
On these bases, protecting the ER via the administration of antioxidants or activation of peroxisome proliferator-activated receptor (PPAR) has been suggested as promising avenues against hepatic steatosis (59, 60)
They play a role in controlling inflammatory reactions and tolerance development [3]
Analysis of the Th cell subset profile in COVID-19 revealed a decline in the percentage of Th17.1 and Th1 lymphocytes capable of producing IFN- (145)
