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6.2.5 Implication for MFH These genetic axes motivate hypotheses that MFH actives could: (i) support mitophagy/mitochondrial quality control, (ii) reinforce endogenous redox buffering (e.g., Nrf2-GPX4), and (iii) dampen inflammation linked to mitochondrial stress

Hennig Frontiers in Immunology.2023;[Epub] CrossRef Molecular Mechanisms in Autoimmune Thyroid Disease Hernando Vargas-Uricoechea Cells.2023